My co-authors and I would like to share the news that several papers resulting from our investigation of the influence of genetic variation on susceptibility of bottlenose dolphins to harmful algal blooms have been published over the past year. Our most recent publication, “Genomewide investigation of adaptation to harmful algal blooms in bottlenose dolphins (Tursiops truncatus),” is now available as Early View online in the journal Molecular Ecology. Cammen, K. M., Schultz, T. F., Rosel, P. E., Wells, R. S. and Read, A. J. (2015), Genomewide investigation of adaptation to harmful algal blooms in common bottlenose dolphins (Tursiops truncatus). Molecular Ecology. doi: 10.1111/mec.13350 Abstract. Harmful algal blooms (HABs), which can be lethal in marine species and cause illness in humans, are increasing worldwide. In the Gulf of Mexico, HABs of *Karenia brevis* produce neurotoxic brevetoxins that cause large-scale marine mortality events. The long history of such blooms, combined with the potentially severe effects of exposure, may have produced a strong selective pressure for evolved resistance. Advances in next-generation sequencing, in particular genotyping-by-sequencing, greatly enable the genomic study of such adaptation in natural populations. We used restriction site-associated DNA (RAD) sequencing to investigate brevetoxicosis resistance in common bottlenose dolphins (*Tursiops truncatus*). To improve our understanding of the epidemiology and aetiology of brevetoxicosis and the potential for evolved resistance in an upper trophic level predator, we sequenced pools of genomic DNA from dolphins sampled from both coastal and estuarine populations in Florida and during multiple HAB-associated mortality events. We sequenced 129 594 RAD loci and analysed 7431 single nucleotide polymorphisms (SNPs). The allele frequencies of many of these polymorphic loci differed significantly between live and dead dolphins. Some loci associated with survival showed patterns suggesting a common genetic-based mechanism of resistance to brevetoxins in bottlenose dolphins along the Gulf coast of Florida, but others suggested regionally specific mechanisms of resistance or reflected differences among HABs. We identified candidate genes that may be the evolutionary target for brevetoxin resistance by searching the dolphin genome for genes adjacent to survival-associated SNPs. Other related papers include: Cammen KM, Wilcox LA, Rosel PE, Wells RS, Read AJ (2015) From genome-wide to candidate gene: An investigation of variation at the major histocompatibility complex in bottlenose dolphins exposed to red tides. Immunogenetics 67:125-133. Cammen KM, Rosel PE, Wells RS, Read AJ (2014) Lack of variation in voltage-gated sodium channels of common bottlenose dolphins exposed to neurotoxic algal blooms. Aquatic Toxicology 157:150-158. All PDFs are available from the respective journals’ websites. Alternatively, pdf requests can be sent to kristina.cammen@maine.edu. -- Kristina Cammen NSF Postdoctoral Fellow in Biology School of Marine Sciences University of Maine, Orono